We’ll start by sharing the following article by Andre Angelantoni: Why do people get the flu after getting the flu shot? He says:
What is actually happening is that the immune system is being programmed by the flu shot making it ready for the exact strains in the shot. It does not protect against other strains but instead opens you up to other flu strains and infection from other pathogens. It’s called heterologous immunity.
The studies
“Heterologous immunity” is a term meaning our immune system can have two opposing responses to infection, depending on the circumstances – it can be stimulated or suppressed.
The phenomenon of immune suppression in certain circumstances has been known for many years – it was first called “original antigenic sin” and in more recent years has been known as “linked epitope suppression”.
https://en.wikipedia.org/wiki/Original_antigenic_sin
Below is a list of studies that investigate this phenomenon as it relates to people getting sick after flu vaccination. We will add more studies as we find them.
Please note that progress in science occurs in tiny steps, each study contributing a small piece of a puzzle. So few papers will come to a sweeping conclusion from which we can get an overall picture—magazine articles are more likely to do that. Also, most medical studies are funded directly or indirectly by the pharmaceutical industry, often with the goal of improving vaccines, so most will recommend vaccination regardless of study findings.
Seasonal Influenza Vaccine and Increased Risk of Pandemic A/H1N1-Related Illness: First Detection of the Association in British Columbia, Canada
https://www.ncbi.nlm.nih.gov/pubmed/20887210
An outbreak investigation in British Columbia during the late spring of 2009 provided the first indication of an unexpected association between receipt of TIV [trivalent inactivated influenza vaccine] and pH1N1 illness. This led to 5 additional studies through the summer 2009 in Canada, each of which corroborated these initial findings.
HETEROLOGOUS IMMUNITY BETWEEN VIRUSES
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2917921/
We propose that heterologous immunity is an important factor in resistance to and variations of human viral infections and that issues of heterologous immunity should be considered in the design of vaccines.
Randomized Controlled Ferret Study to Assess the Direct Impact of 2008–09 Trivalent Inactivated Influenza Vaccine on A(H1N1)pdm09 Disease Risk
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3903544
In summary, although these ferret findings cannot be considered conclusive in explaining earlier human observations from Canada, they support the hypothesis that prior receipt of 2008–09 TIV may have had direct, adverse effects on A(H1N1)pdm09 illness.
Increased Risk of Noninfluenza Respiratory Virus Infections Associated With Receipt of Inactivated Influenza Vaccine
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3404712/
We identified a statistically significant increased risk of noninfluenza respiratory virus infection among TIV [trivalent inactivated influenza vaccine] recipients (Table 3), including significant increases in the risk of rhinovirus and coxsackie/echovirus infection…
Reduced Antibody Responses to the Pandemic (H1N1) 2009 Vaccine after Recent Seasonal Influenza Vaccination
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3165229/
Following 2009 H1N1 vaccination, subjects previously given a seasonal influenza virus vaccination exhibited significantly lower antibody responses, as determined by hemagglutination inhibition assay, than subjects who had not received the seasonal influenza virus vaccination. This result is compatible with the phenomenon of “original antigenic sin,” by which previous influenza virus vaccination hampers induction of immunity against a new variant. Our finding should be taken into account for future vaccination programs against pandemic influenza virus outbreaks.
Original Antigenic Sin: How First Exposure Shapes Lifelong Anti–Influenza Virus Immune Responses
https://www.jimmunol.org/content/202/2/335
The “negative interference” predicted by the antigenic distance hypothesis was proposed as an explanation for the unusual observation that vaccine efficacy against H3N2 was apparently reduced in repeat vaccinees when compared with first-time vaccinees during the 2012–2013 influenza season in Canada (41)
Influenza Vaccination and Respiratory Virus Interference Among Department of Defense Personnel During the 2017-2018 Influenza Season
https://pubmed.ncbi.nlm.nih.gov/31607599/
Vaccine derived virus interference was significantly associated with coronavirus and human metapneumovirus; however, significant protection with vaccination was associated not only with most influenza viruses, but also parainfluenza, RSV, and non-influenza virus coinfections.
Association between the 2008–09 Seasonal Influenza Vaccine and Pandemic H1N1 Illness during Spring–Summer 2009: Four Observational Studies from Canada
https://journals.plos.org/plosmedicine/article?id=10.1371/journal.pmed.1000258
Prior receipt of 2008–09 TIV was associated with increased risk of medically attended pH1N1 illness during the spring–summer 2009 in Canada.
Epidemiology of respiratory viral infections in children enrolled in a study of influenza vaccine effectiveness
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4181477/pdf/irv0008-0293.pdf
We did, however, unexpectedly find that non-influenza ILI [influenza-like illness] occurred about 1.6 times more commonly in children vaccinated with one or two doses of the influenza vaccine than in unvaccinated children. These results support the findings of a recent RCT (randomized controlled trial) reported by Cowling et al. (increaed risk paper)
Vaccination against Human Influenza A/H3N2 Virus Prevents the Induction of Heterosubtypic Immunity against Lethal Infection with Avian Influenza A/H5N1 Virus
http://vaccinepapers.org/wp-content/uploads/Vaccination-against-Human-Influenza-AH3N2-Virus-Prevents-the-Induction-of-Heterosubtypic-Immunity-against-Lethal-Infection-with-Avian-Influenza-AH5N1-Virus.pdf
Here we show in a mouse model that the induction of protective heterosubtypic immunity by infection with a human A/ H3N2 influenza virus is prevented by effective vaccination against the A/H3N2 strain. Consequently, vaccinated mice were no longer protected against a lethal infection with an avian A/H5N1 influenza virus.
Effectiveness and Cost-Benefit of Influenza Vaccination of Healthy Working Adults
https://jamanetwork.com/journals/jama/fullarticle/193139
In years in which there is a good match between vaccine and circulating viruses, vaccination against influenza can have substantial health benefits by reducing rates of ILI [influenza-like illness], physician visits, and work absenteeism. Nonetheless, our results suggest that vaccination of healthy adults younger than 65 years is unlikely to provide societal economic benefit in most years.
Assessment of temporally-related acute respiratory illness following influenza vaccination
https://www.sciencedirect.com/science/article/pii/S0264410X18303153
Patient perceptions of illness following influenza vaccination may be supported. … Among children there was an increase in the hazard of ARI [acute respiratory illness] caused by non-influenza respiratory pathogens post-influenza vaccination compared to unvaccinated children during the same period. Potential mechanisms for this association warrant further investigation.
– Note that the follow-up period in this study was only 14 days after vaccination.
COVID-19
COVID-19 Severity in Europe and the USA: Could the Seasonal Influenza Vaccination Play a Role?
https://pubmed.ncbi.nlm.nih.gov/32714123/
Our analysis indicates that receiving seasonal influenza vaccination(s) in the past might be an additional risk factor for the elderly in terms of enhanced susceptibility to infection with SARS-CoV-2 and higher likelihood of a lethal outcome in case of infection. More research about this possible risk factor is urgently needed.
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The following 2 studies when taken together suggest Flu vaccines would inhibit the recipient’s immune reaction to the virus SARS-CoV-2, which causes Coronavirus Disease 19 i.e. COVID-19. The first paper, from Melbourne, shows a COVID-19 patient developed CD8+ T cells prior to recovery, while the second paper found flu vaccine hampered production of CD8+ T cells.
Breadth of concomitant immune responses prior to patient recovery: a case report of non-severe COVID-19
https://www.nature.com/articles/s41591-020-0819-2
We report the kinetics of immune responses in relation to clinical and virological features of a patient with mild-to-moderate coronavirus disease 2019 (COVID-19) that required hospitalization. Increased antibody-secreting cells (ASCs), follicular helper T cells (TFH cells), activated CD4+ T cells and CD8+ T cells and immunoglobulin M (IgM) and IgG antibodies that bound the COVID-19-causing coronavirus SARS-CoV-2 were detected in blood before symptomatic recovery.
Annual Vaccination against Influenza Virus Hampers Development of Virus-Specific CD8+ T Cell Immunity in Children
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3209321/
Our results indicate that annual influenza vaccination is effective against seasonal influenza but hampers the development of virus-specific CD8+ T cell responses. The consequences of these findings are discussed in the light of the development of protective immunity to seasonal and future pandemic influenza viruses.
Articles
Yearly influenza vaccinations: a double-edged sword?
Yearly vaccination against seasonal influenza viruses is recommended for certain individuals at high risk of complications associated with influenza. … However, it has been shown—mainly in animals—that infection with influenza A viruses can induce protective immunity to influenza A viruses of other unrelated subtypes. … This type of immunity might be relevant to human beings when a new subtype of influenza A virus is introduced into the population … Preventing infection with seasonal influenza viruses by vaccination might prevent the induction of heterosubtypic immunity to pandemic strains, which might be a disadvantage to immunologically naive people—eg, infants.
Pentagon Study: Flu Shot Raises Risk of Coronavirus by 36% (and Other Supporting Studies)
Find a collection of studies showing Flu vaccines increase infections.
Influenza Vaccine Causes Illness and Immune Dysfunction
A poorly-matched influenza vaccine may cause illness (and increase risk of influenza illness) by a phenomenon in immunology known as “original antigenic sin” (OAS). First discovered in 1960, OAS is well known and firmly established. Its described in any immunology textbook. … By receiving an influenza vaccine that is poorly matched to circulating strains, the immune system is improperly trained. Improper immune training is worse than no training at all.
Vaccination may make flu worse if exposed to a second strain
A new study in the U.S. has shown that pigs vaccinated against one strain of influenza were worse off if subsequently infected by a related strain of the virus.
Melbourne Medical Doctor And Researcher Professor Frank Shann Believes Non-Live Vaccines Increase Infant Deaths In Third-World Countries
Frank Shann, Peter Aaby and other scientists have found non-live vaccines in general increase off-target infections – article on this website.
Andre Angelantoni’s article again:
Why do people get the flu after getting the flu shot?
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